AMPAR

基本解釋

In vivo treatment with AMPAR antagonists blocked increases in phosphorylation, EPSC amplitude, and EPSC frequency, and also attenuated susceptibility to later seizures.
在活體治療中阻斷AMPAR拮抗劑可增加磷酸化，增加EPSC振幅與頻率，同時(shí)也減輕了后天癲癇發(fā)作的損傷。
The distribution and match relationship of the 4 subunits of AMPA receptor (AMPAR) and glutamate (Glu) in the caudal trigeminal subnucleus (Vc) of rat were immunohistochemically investigated by using ABC method.
本文用免疫組織化學(xué)ABC法調(diào)查了4種AMPA受體亞單位（GluR1、2/3、4）和谷氨酸（Glu）在大鼠三叉神經(jīng)尾側(cè)亞核（Vc）內(nèi)的分布及其匹配關(guān)系。
This increase likely resulted from increased phosphorylation of AMPAR subunits by PKA, PKC, and calcium/calmodulin-dependent kinase II, because the activity of these kinases was elevated.
這可能是由于PKA,PKC,以及鈣/鈣依賴性激酶II使AMPAR亞基磷酸化增多，因?yàn)榇藭r(shí)這些酶的活性也提高了。
Čampar
錢帕爾